© 2002 by Heart
VIEWPOINT
Vasomotion and nitric oxide bioactivity in diseased coronary arteries
1 Cardiology Unit, Athens University Medical School, Athens, Greece
2 Division of Cardiology, Hammersmith Hospital, London, UK
3 Department of Cardiology, St Bartholemew's Hospital, London, UK
Correspondence to:
Correspondence to:
Graham J Davies, Division of Cardiology, Hammersmith Hospital, Du Cane Road, London W12 ONN, UK;
gjdavies{at}hhnt.org
Atheromatous coronary stenoses are no longer considered to be passive structures, instead having the capacity for dynamic, often transient, change which may take the form of constriction or dilatation in response to either endogenous or external stimulation.
Keywords: vasomotion; nitric oxide; coronary artery disease
Abbreviations: iNOS, cytokine induced nitric oxide synthase; LNMMA, N-monomethyl L-arginine
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