Editorial
Does smoking cessation reduce the risk of restenosis following coronary angioplasty?
| The first 150 words of the full text of this article appear below. |
Before the widespread use of coronary stents, restenosis occurred in around one third of lesions dilated by percutaneous transluminal coronary angioplasty (PTCA).1 The actual rates reported in individual case series vary widely depending on patient selection, the definition of restenosis applied, duration of follow up, completeness of angiographic follow up, and whether or not quantitative methods of assessment are used.
Although infrequently resulting in myocardial infarction or death,1 2 restenosis is nonetheless an important occurrence since it is associated with the recurrence or deterioration of symptoms and, in some patients, necessitates further attempts at revascularisation.
PTCA does not prevent continued progression of the underlying
atherosclerotic disease which may result in further stenoses both at
the dilatation site and elsewhere in the coronary arterial system.
Furthermore, use of balloon dilatation has been implicated as
increasing the risk of early restenosis through both direct and
indirect effects. Direct intimal injury at or near
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Tobacco use
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Drugs: cardiovascular system
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Acute coronary syndromes
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Interventional cardiology
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