Editorial
Prevention of contrast nephropathy after cardiac catheterisation
| The first 150 words of the full text of this article appear below. |
Increasing numbers of patients in the developed world are exposed to contrast medium during cardiac catheterisation procedures, and the pressure to increase cardiac laboratory throughput of patients with suspected coronary disease is unrelenting. For this reason, the problem of radiocontrast induced nephropathy (RCIN) assumes greater and greater importance. How is the fallout from contrast exposure to be minimised?
Scale of the problemRadiocontrast use in all branches of medicine is reported to be
the third most common cause of new onset renal failure in hospital
patients.1 Although varying definitions of nephropathy have been employed in the literature, including a 25% rise, a 50%
rise or a doubling of serum creatinine concentration, it is clear that
this is a common complication of cardiac catheterisation. In the recent
epidemiological report by McCullough and colleagues, 1826 unselected
patients undergoing coronary intervention were found to have a 14.5%
incidence of acute contrast induced renal failure
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