© 2002 by Heart
EDITORIAL
The right atrium as an anatomic set-up for re-entry: electrophysiology goes back to anatomy
Correspondence to:
Correspondence to:
Dr Francisco G Cosío, Cardiology Service, Hospital Universitario de Getafe, Carretera de Toledo, km 12.5, 28905 Getafe, Madrid, Spain;
fcosio@vitanet.nu
Rapid, complete, uniform activation of all atrial tissue is important for rhythm stability. Typical flutter, the most common among atrial tachycardias, has anisotropic conduction at the terminal crest as an essential part of its mechanism
Keywords: right atrium; terminal crest; typical flutter; myocardial anisotropy
| The first 150 words of the full text of this article appear below. |
Normal heart activation is characterised by regular alternation between depolarisation/repolarisation and rest. Activation originates in the right atrium, around the sinus node area, and spreads until the atria are completely activated. Once activation is complete atrial tissue is refractory and a period of rest necessarily precedes the next activation cycle. This sequence is caused by pacemaker cells with discharge rates that respond to neural and humoral stimuli, thus allowing adaptation of heart function to physiologic demands. However, this normal sequence is not so simple to sustain, as demonstrated by the frequent appearance of re-entrant tachycardias.
Rapid, complete, uniform activation of all atrial tissue is important for rhythm stability. Preferential conduction pathways have been long recognised in the atria,1–3 despite the absence of bundles of specialised conduction akin to the His-Purkinje network of the ventricles.4 James and Sherf5 attributed the faster conduction along the terminal crest and Bachmann’s bundle to the
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