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LettersThe authors’ reply:
1 University of Sydney, Westmead Hospital, Sydney, Australia
2 University of Queensland, Princess Alexandra Hospital, Brisbane, Australia
3 University of NSW at Liverpool and University of Sydney, Sydney, Australia
Correspondence to:
Correspondence to Dr Suzanne Eshoo, Department of Cardiology, Westmead Hospital, Darcy Road, Westmead, 2145, NSW, Australia; suzanne.eshoo@sswahs.nsw.gov.au
| The first 150 words of the full text of this article appear below. |
We appreciate the interest of Zhang and co-workers in our article1 and are grateful for the opportunity to respond to the issues raised.
We appreciate that diabetes mellitus and insulin resistance have an independent effect on diastolic function even in the absence of hypertension.2 In our study, in fact only 14% of the patients had diabetes mellitus—that is, seven patients of 54, not 14/54 as stated. In order to demonstrate that these seven diabetic patients had not confounded our results, analysis was repeated after exclusion of the seven diabetic patients; maximal LA volume showed no difference between groups with a decrease in indexed conduit volume (11.1 (SD 7.2) ml/m2 vs 13.8 (6.1) ml/m2, p = 0.04). E-Sr remained decreased in the hypertension group compared to normals (1.9 (0.6) s–1 vs 2.2 (0.7) s–1, p = 0.02). There was no difference in global S-Sr, A-Sr or global strain.
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