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Heart 2009;95:1628-1634; doi:10.1136/hrt.2008.151175
Copyright © 2009 BMJ Publishing Group Ltd & British Cardiovascular Society

Education in Heart

Congenital heart disease

Management of asymptomatic Wolff–Parkinson–White syndrome

John K Triedman

Correspondence to:
Correspondence to Dr John Triedman, Children’s Hospital Boston, Harvard Medical School, 300 Longwood Avenue, Boston, MA 02115, USA; john.triedman@cardio.chboston.org

The first 150 words of the full text of this article appear below.

Wolff–Parkinson–White syndrome (WPW) is a frequently encountered electrocardiographic abnormality and an important diagnostic and therapeutic issue for cardiac specialists. Estimates of WPW prevalence in the general population are contingent on the likelihood of asymptomatic patients having an electrocardiogram (ECG) performed, and range from 0.1–0.2%. WPW consists of pre-excitation of the QRS (the delta wave) caused by eccentric activation of the ventricular myocardium via an accessory atrioventricular (AV) connection (accessory pathway, historically called the bundle of Kent) (fig 1). In addition to ventricular pre-excitation observed in sinus rhythm, the electrophysiological consequences of this connection include the paroxysmal occurrence of atrioventricular reciprocating tachycardia (SVT) and, at considerably lower frequency, the occurrence of rapidly conducted atrial fibrillation, which may result in ventricular fibrillation and death.


 

Aetiology and pathophysiology

Accessory pathways (APs) by definition traverse the AV groove, and are identified in all anatomical quadrants of the . . . [Full text of this article]


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