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Heart 2009;95:1803-1807; doi:10.1136/hrt.2008.160382
Copyright © 2009 BMJ Publishing Group Ltd & British Cardiovascular Society

Education in Heart

Arrhythmias

Drug treatment of supraventricular tachycardia

Carina Blomström-Lundqvist

Correspondence to:
Correspondence to Professor Carina Blomström-Lundqvist, Department of Cardiology, University Hospital, Uppsala University, Uppsala, Sweden; carina.blomstrom.lundqvist@akademiska.se

The first 150 words of the full text of this article appear below.

Supraventricular tachycardia (SVT) is characterised by a rapid impulse formation, that emanates from the sinus node, from atrial tissue (focal or macro-reentrant atrial tachycardia (AT)), from the atrioventricular (AV) node, or from anomalous muscle fibres that connect the atrium with the ventricle (accessory pathways (APs)). The most frequently (90%) encountered SVTs are AV nodal reentrant tachycardia (AVNRT), AV reentrant tachycardia (AVRT) mediated by accessory pathways, and atrial flutter (AFL). The remaining SVTs are AT and non-paroxysmal, usually incessant, forms of SVT.

Paroxysmal forms of SVT (PSVT) are regular recurrent tachycardias with a sudden onset and termination. If terminated by vagal manoeuvres, a reentrant tachycardia involving the AV node is most likely. The ventricular rate during SVT is commonly between 140–250 beats/min (bpm). If vagal or pharmacologic manoeuvres (adenosine) during an SVT result in AV block with persistence of atrial tachycardia, the diagnosis is most likely AT. The A:V ratio . . . [Full text of this article]


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