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Editorial
Idiopathic ventricular outflow tract tachycardia
  1. A Farzaneh-Far,
  2. B B Lerman
  1. Department of Medicine, The Maurice & Corinne Greenberg Division of Cardiology, Cornell University Medical Center, New York, USA
  1. Correspondence to:
    Bruce B Lerman
    Department of Medicine, Division of Cardiology, Cornell University Medical Center, 525 East 68th Street, Starr 409, New York, NY 10021, USA; blermanmed.cornell.edu

Abstract

Although the pathogenesis of ventricular outflow tract tachycardia has not been fully elucidated, recent findings suggest that defects in cAMP signalling may be involved

  • ARVD, arrhythmogenic right ventricular dysplasia
  • cAMP, cyclic adenosine monophosphate
  • DAD, delayed afterdepolarisation
  • GTP, guanosine triphosphate
  • MRI, magnetic resonance imaging
  • RVOT, right ventricular outflow tract
  • VT, ventricular tachyarrhythmia
  • adenosine
  • triggered activity
  • ventricular tachycardia

https://doi.org/10.1136/hrt.2004.033795

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