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Impaired left ventricular energy metabolism in hypertrophic cardiomyopathy is not due to fibrosis
  1. S E Petersen,
  2. H Watkins,
  3. S Neubauer
  1. Department of Cardiovascular Medicine, University of Oxford, John Radcliffe Hospital, Oxford, UK
  1. Dr S E Petersen, Department of Cardiovascular Medicine, University of Oxford, John Radcliffe Hospital, Oxford OX3 9DU, UK; steffen.petersen{at}cardiov.ox.ac.uk

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To the editor: Esposito et al deal with an important pathophysiological question—that is, whether altered cardiac energy metabolism in hypertrophic cardiomyopathy (HCM) is primary or secondary, due to fibrosis.1 The authors conclude that the inverse relation seen between LE extension (LE, late enhancement a measure of fibrosis) and …

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