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  • YIA5 PKCδ deficiency accelerates neointimal lesions in a mouse model of vascular injury involving delayed re-endothelialisation and vasohibin-1 accumulation

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BSCR Spring 2010 Meeting Abstracts
BAS/BSCR YIA abstract
YIA5 PKCδ deficiency accelerates neointimal lesions in a mouse model of vascular injury involving delayed re-endothelialisation and vasohibin-1 accumulation
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Cite this article as:
Bai X, Margariti A, Zeng L, et al
YIA5 PKCδ deficiency accelerates neointimal lesions in a mouse model of vascular injury involving delayed re-endothelialisation and vasohibin-1 accumulation
Heart 2010;96:e10.