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BSCR Autumn Meeting Abstracts, 2009
Myocardial energetics and redox in health and disease
008 Neuronal nitric oxide synthase-mediated regulation of myocardial redox state and β3 adrenergic receptor responses
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  1. W Idigo
  1. Department of Cardiovascular Medicine, University of Oxford, John Radcliffe Hospital, Oxford, UK

https://doi.org/10.1136/hrt.2009.191049h

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    Recent evidence suggests that increased superoxide production may decrease the bioavailability of endothelial nitric oxide synthase (eNOS)-derived nitric oxide in the myocardium of neuronal nitric oxide synthase (nNOS)−/− mice. To test the functional relevance of this observation, we compared the eNOS-dependent inotropic and [Ca2+]i transient effect of beta3-adrenergic receptor (β3-AR) stimulation in left ventricular myocytes from eNOS …

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