- Hepatocyte growth factor
- heart failure
- cardiac remodelling
- left ventricular hypertrophy
- aortic valve disease
- gene expression
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KR and BR contributed equally to this study.
Funding This work was supported by the Association for the Promotion of Research in Arteriosclerosis, Thrombosis and Vascular Biology (Vienna, Austria) and by the Ludwig Boltzmann Foundation for Cardiovascular Research (Vienna, Austria).
Competing interests None.
Patient consent Obtained.
Ethics approval This study was approved by the ethics committee of the Medical University of Vienna, Austria.
Provenance and peer review Not commissioned; externally peer reviewed.
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