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To the Editor Tumour necrosis factor-α (TNF-α) has been implicated in the development of acute myocardial infarction. The mechanisms of TNF-α on endothelial function and platelet activation in patients with acute myocardial infarction are still unclear. A recent article by Padfield et al1 demonstrated that TNF-α antagonism is unlikely to be a beneficial therapeutic strategy in patients with acute myocardial infarction. And plasma TNF-α concentrations increased in all patients following etanercept. This …
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