Biochemical and Biophysical Research Communications
Regular ArticleConstruction of a Human Cardiovascular cDNA Microarray: Portrait of the Failing Heart
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2019, Pharmacological ResearchCitation Excerpt :In addition, to evaluate the specificity of these 608 immunological signatures for immune systems, we inspected the involvement of the immunological signatures in seven gene expression profiles of different cardiomyopathy subtypes from a same experiment (GEO accession: GSE2656; Supplementary table 2). This experiment utilizes a homemade cDNA microarray (termed CardioChip; GEO accession: GPL2041), containing 10,368 sequence tags specifically derived from human heart and artery cDNA libraries and applied to detecting the portrait of the failing heart, thus, does not provides adequate coverage on the immune systems related genes [14]. For the 7 heart diseases, we would expect that the 608 immunological signatures have no significant associations with their transcriptional profiles (FDR > 0.05).
Association of expression of ZNF606 gene from monocytes with the risk of coronary artery disease
2018, Clinical BiochemistryCitation Excerpt :As a result, monocytes may act as a potential target for diagnosing the risk associated with CAD and as a promising therapy. Next-generation sequencing and high-density microarrays have been established as novel methods for CAD study [32], which provides us with more information regarding the changes of gene expression during the disease development. ZNF606 is a zinc finger protein that contains a Kruppel-associated box domain (KRAB) in the N-terminal end, and functions as a transcriptional repression domain.
Overexpression of prostaglandin E2 EP4 receptor improves cardiac function after myocardial infarction
2018, Journal of Molecular and Cellular CardiologyMolecular distinction between physiological and pathological cardiac hypertrophy: Experimental findings and therapeutic strategies
2010, Pharmacology and TherapeuticsCitation Excerpt :The enhanced oxidative capacity of hearts that have undergone physiological hypertrophy suggests a protected phenotype. In another approach to identify transcriptome changes associated with pathological and physiological cardiac hypertrophy, a number of investigators have conducted comprehensive microarray gene expression profiling studies in hearts from rodent models of pathological and physiological cardiac hypertrophy, as well as tissue from heart failure patients (Friddle et al., 2000; Hwang et al., 2000; Yang et al., 2000; Barrans et al., 2001; Hwang et al., 2002b; Diffee et al., 2003; McMullen et al., 2004b; Kong et al., 2005; Strom et al., 2005). More recent studies have used proteomic methods, such as 2-dimensional polyacrylamide gel electrophoresis (2-D PAGE) in conjunction with mass spectrometry to catalogue exercise-induced changes in the cardiac proteome (Boluyt et al., 2006; Burniston, 2009).
Transcriptomics: Translation of Global Expression Analysis to Genomic Medicine
2009, Genomic and Personalized Medicine, Two-Vol SetTranscriptomics: Translation of Global Expression Analysis to Genomic Medicine
2008, Genomic and Personalized Medicine: V1-2