Coronary heart disease: Epidemiology of smoking and intervention studies of smoking☆
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Effect of smoking on culprit lesion plaque burden and composition in acute coronary syndrome: An intravascular ultrasound-virtual histology study
2021, Indian Heart JournalCitation Excerpt :Impairment of vasomotor function, inflammation and oxidation of lipids are major mechanisms for initiation and progression of atherosclerosis which appear prior to the onset of structural and pathologic manifestations.3–7 Smoking mainly leads to acute coronary events and less for the occurrence of stable angina.8 Further, studies demonstrated a faster reversal in cardiovascular risk following smoking cessation as compared to reduction seen with cholesterol and blood pressure lowering drugs, which predominantly target atherosclerotic process.9–11
Activation of NLRP3 inflammasome assembly is associated with smoking status of patients with coronary artery disease
2020, International ImmunopharmacologyCitation Excerpt :The imperative determinants of atherogenesis embrace the concomitant risk factors such as smoking, hypercholesterolemia, etc. [1]. Smoking is one of the major, modifiable and well-known risk factors that contribute to atherosclerotic progression [1,2]. Smoking is known to induce oxidative stress that may perpetuate a pro-inflammatory response [1].
Tobacco and arteritis
2016, Archives des Maladies du Coeur et des Vaisseaux - PratiqueAssociation between smoking habits and the first-time appearance of atrial fibrillation in Japanese patients: Evidence from the Shinken Database
2015, Journal of CardiologyCitation Excerpt :Tobacco smoking is a well-known risk factor for cardiovascular disease [20,21]. Smoking causes endothelial dysfunction and atherosclerosis [22–25], and also can cause cardiac arrhythmias [26] through the combined effects of nicotine, carbon monoxide, and polycyclic aromatic hydrocarbons. These studies indicate that tobacco smoking may accelerate atrial remodeling, contributing to the development of AF.
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Supported by grants from The Swedish National Bank Tercentenary Fund, The Swedish Association against Heart and Chest Diseases, and The Knut and Alice Wallenberg Foundation.