Original article: CardiovascularCirculating endothelin in cardiac operations: influence of blood pressure and endotoxin
References (31)
- et al.
Serum endothelin-1 concentrations and cold provocation in primary Raynaud's phenomenon
Lancet
(1990) - et al.
Hemodynamic shear stress stimulates endothelin production by cultured endothelial cells
Biochem Biophys Res Commun
(1989) - et al.
Endotoxin stimulates endothelin release in vivo and in vitro as determined bv radio-immunoassay
Biochem Biophys Res Commun
(1989) - et al.
Increased plasma and pulmonary lymph levels of endothelin during endotoxin shock
Eur J Pharmacol
(1989) - et al.
Increased plasma levels of endothelin-1 after cardiopulmonary bypass in patients with pulmonary hypertension and congenital heart disease
J Thorac Cardiovasc Surg
(1993) - et al.
Endotoxin release and tumor necrosis factor formation during cardiopulmonary bypass
Ann Thorac Surg
(1992) - et al.
Endotoxemia associated with cardiopulmonary bypass
J Thorac Cardiovasc Surg
(1987) - et al.
Presence of circulating endotoxins during cardiac operations
J Thorac Cardiovasc Surg
(1987) - et al.
The effect of intestinal hypoperfusion on intestinal absorption and permeability during cardiopulmonary bypass
Gastroenterology
(1994) - et al.
Increased plasma endothelin immunoreactivity during cardiopulmonary bypass: a preliminary observation
J Thorac Cardiovasc Surg
(1992)
Role of endothelin 1 in hemorhagic shock-induced gastric mucosal injury in rats
Gastroenterology
(1994)
A novel potent vasoconstrictor peptide produced by vascular endothelial cells
Nature
(1988)
Endothelin: a novel peptide in the posterior pituitary system
Science
(1989)
Endothelin, a potent vasoconstrictor, is a vascular smooth muscle mitogen
J Vasc Biol Med
(1989)
Plasma endothelin during upright tilt: relevance for orthostatic hypotension?
Lancet
(1991)
Cited by (27)
Natural History of Saphenous Vein Grafts
2013, Interventional Cardiology ClinicsCitation Excerpt :In addition, saphenous veins, particularly when denuded, are highly sensitive to circulating vasoconstrictors, including the most potent endogenous vasoconstrictor, endothelin-1. The circulating concentration of endothelin-1 shows a marked initial increase followed by an additional slower increment, after the onset of cardiopulmonary bypass,16 and the resulting venoconstrictor response may further attenuate flow and promote stasis. In SVGs, the predominant vasomotor response to thrombin is a constrictor one, in contrast to the thrombin-mediated vasorelaxation that occurs via endothelial receptors in internal mammary arteries, further promoting thrombosis in SVGs.17
Regional Circulation
2011, Pediatric Critical Care: Expert Consult Premium EditionRegional Circulation
2011, Pediatric Critical CareCharacterizing the Inflammatory Response to Cardiopulmonary Bypass in Children
2006, Annals of Thoracic SurgeryRegional Circulation
2006, Pediatric Critical Care
Copyright © 1996 Published by Elsevier Inc.