Exercise-induced left ventricular dysfunction in alcoholic and non-alcoholic cirrhosis
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2021, JACC: Basic to Translational ScienceCitation Excerpt :The exact mechanism of the increased cardiac output in cirrhosis at rest is not well understood, but it may be related to reduced central and arterial blood volume in patients with cirrhosis (74), which would lead to deactivation of arterial baroreceptors and an increase in the activity of the sympathetic nervous system and thus an increase in heart rate and cardiac output. The reduced cardiac performance in cirrhosis during stress may relate to blunted heart rate response, reduced myocardial contractility, and significant wasting of skeletal muscles (74,76-78). Furthermore, cirrhosis is associated with dysfunctional plasma membrane and ion channels, which predisposes patients with cirrhosis to conduction abnormalities and arrhythmias, which can further exacerbate cardiac dysfunction (74).
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2018, Ultrasound in Medicine and BiologyCitation Excerpt :Moreover, the use of a multi-parameter approach in the DD quantification algorithm allows us to better classify the severity of DD and, importantly, to estimate LV filling pressure in LC patients in the context of high pre-load conditions. No correlation was found between the presence of DD and alcoholic or non-alcoholic etiology, in agreement with other studies (Bernardi et al. 1998; Grose et al. 1995; Pozzi et al. 1997). This suggests that LC per se generates cardiac dysfunction.