Table 2 Patient demographic and clinical characteristics
VariablesEarly Gp IIb–IIIa inhibitorsLate Gp IIb–IIIa inhibitors
(n  =  840)(n  =  822)
Age, years
    Median6161
    Range52–7052–70
Sex, n (%)642/840 (76.4%)641/822 (78.0%)
Hypertension, n (%)353/838 (42.1%)347/822 (42.2%)
Diabetes, n (%)123/840 (14.6%)135/822 (16.4%)
Previous MI, n (%)67/838 (8.0%)80/822 (9.7%)
Previous revascularisation, n (%)61/792 (7.7%)59/770 (7.7%)
Smoking, n (%)440/840 (52.4%)419/822 (51.0%)
Hypercholesterolemia, n (%)298/840 (35.5%)309/820 (37.7%)
Killip class III/IV, n (%)33/722 (4.6%)33/705 (4.7%)
Anterior MI, n (%)361/831 (43.4%)369/819 (45.1%)
Symptom onset to Gp IIb–IIIa inhibitor time, minutes*
    Median100197
    25–75th percentiles65–178144–275
Ischaemia time, minutes
    Median193203
    25–75th percentiles146–270150–285
Infarct-related artery
    LAD, n (%)351(41.7%)361 (43.9%)
    CX, n (%)124 (14.7%)100 (12.1%)
    RCA, n (%)339 (40.3%)336 (40.9%)
    GRAFT, n (%)6 (0.7%)6 (0.7%)
    LM, n (%)4 (0.5%)5 (0.6%)
Multivessel disease, n (%)437/757 (57.7%)433/737 (58.8%)
Follow-up
    Median330347
    25–75th percentiles30–36030–360
  • All p values are not significant except for the time from symptom onset to administration of Gp IIb–IIIa inhibitors* (p<0.001).

  • CX, circumflex artery; Gp, glycoprotein; LAD, left descending coronary artery; LM, left main artery; MI, myocardial infarction; RCA, right coronary artery.