Arrhythmias and conduction disturbanceRelation between site of origin of ventricular tachycardia and relative left ventricular myocardial perfusion and wall motion☆
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Role of Cardiac Imaging in Evaluating Risk for Sudden Cardiac Death
2017, Cardiac Electrophysiology ClinicsCitation Excerpt :The presence and size of resting perfusion defects in patients with ischemic cardiomyopathy (ICM) have been shown to correlate with the inducibility of ventricular arrhythmias during an electrophysiologic study.75,76 VTs were found to originate close to the border of the resting perfusion defects.77 Moreover, it was demonstrated that impairment of thallium uptake, signifying zone of infarct, accurately predicted endocardial voltage-defined scar, which was the site of successful ablation in all patients.78
Localization of Ventricular Tachycardia Exit Site and Subsequent Contraction Sequence and Functional Effects With Bedside Radionuclide Angiography
2008, JACC: Cardiovascular ImagingCitation Excerpt :Aneurysms have previously been shown to appear with pacing of the peri-infarction area in animals (14) and to resolve with resynchronization therapy (14,21), based likely on altered conduction (21). When using electrophysiologic studies, researchers often have localized the VT exit site to the perianeurysmal border region (5), which also was confirmed in our study, as ERNA often demonstrated new aneurysms with VT. The study population was small in this challenging protocol but adequately served the purpose.
Relationship of scar and ischemia to the results of programmed electrophysiological stimulation in patients with coronary artery disease
1997, Journal of Nuclear CardiologyVentricular tachycardia after infarction: Sources of coronary blood flow to the infarct zone
1992, American Heart JournalPrevalence of ischemia by quantitative thallium-201 scintigraphy in patients with ventricular tachycardia or fibrillation inducible by programmed stimulation
1987, The American Journal of CardiologyLong-term reproducibility of responses to programmed cardiac stimulation in spontaneous ventricular tachyarrhythmias
1984, The American Journal of Cardiology
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This study was supported in part by a grant (R01 HL24278) from the National Heart, Lung, and Blood Institute, Bethesda, Maryland, and a grant from the American Heart Association, Southeastern Pennsylvania Chapter, Philadelphia, Pennsylvania.
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Dr. Josephson is the Robinette Foundation Associate Professor of Medicine (Cardiovascular Diseases) and the recipient of a Research Career Development Award (K04 HL00361) from the National Heart, Lung, and Blood Institute, Bethesda, Maryland.