Platelet hyperaggregability in patients with chest pain and angiographically normal coronary arteries
References (33)
- et al.
Variant angina: one aspect of a continuous spectrum of vasospastic myocardial ischemia
Am J Cardiol
(1978) Elevated left ventricular filling pressures as a cause of chest pain in patients with normal coronary arteries (abstr)
Am J Cardiol
(1982)- et al.
Unimproved chest pain in patients with minimal or no coronary disease: a behavioral phenomenon
Am Heart J
(1984) - et al.
Adenosine diphosphate induced platelet aggregation in myocardial infarction and ischemic heart disease
Atherosclerosis
(1973) - et al.
Platelet function studies in coronary artery disease
Am J Cardiol
(1979) - et al.
Plasma heparin neutralizing activity in coronary artery disease
Am J Cardiol
(1976) - et al.
Elevated beta thromboglobulin in peripheral venous blood of patients with acute myocardial ischemia: direct evidence for enhanced platelet reactivity in vivo
Am J Cardiol
(1981) - et al.
Exercise-induced myocardial ischemia in patients with coronary artery disease: lack of evidence for platelet activation or fibrin formation in peripheral venous blood
JACC
(1983) - et al.
Platelet function studies in coronary artery disease
Am J Cardiol
(1980) Effects induced on blood platelets in ischemic and nonischemic myocardium
Am J Cardiol
(1983)
Comparison of platelet function during exercise in normal subjects and coronary artery disease patients: potential role of platelet activation in myocardial ischemia
Am Heart J
Intravascular microaggregation and in vitro platelet aggregation in coronary artery disease
Thromb Res
Platelet activation in clinical coronary artery disease and spasm
Am Heart J
The anginal syndrome with normal coronary arteriography
Trans Assoc Am Physicians
Paradox of normal selective coronary arteriograms in patients considered to have unmistakable coronary heart disease
N Engl J Med
The anginal syndrome associated with normal coronary arteriograms
Am J Med
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Sticky platelet syndrome. A report of 2 cases
2016, Revista Espanola de Cirugia Oral y Maxilofacial<sup>1</sup>H NMR-derived metabolomics of filtered serum of myocardial ischemia in unstable angina patients
2016, Clinica Chimica ActaCitation Excerpt :Moreover, not only the reduced levels of adenosine, hypoxanthine, xanthine and inosine in serum can be a manifestation of their increased urinary excretion, which is established as urinary markers of cardiac ischemia [35] but also exogenous inosine administration has been evidenced for cardioprotection [36]. The correlation between platelet aggregation and CAD has well been documented in various studies [37,38]. Adenosine diphosphate (ADP) is a known agonist for platelet aggregation [39,40].
Increased soluble platelet/endothelial cell adhesion molecule-1 in the early stages of acute coronary syndromes
2003, International Journal of CardiologySticky platelet syndrome and thrombocythemia
2003, Hematology/Oncology Clinics of North AmericaElevated soluble P-selectin levels are associated with an increased risk of early adverse events in patients with presumed myocardial ischemia
2002, American heart journalCitation Excerpt :Gurbel et al31 have also recently reported elevated sP-selectin levels in patients with noncardiac chest pain. Likewise, patients with chest pain but with angiographically normal coronary arteries exhibit other features of increased platelet activity,33 whereas sP-selectin levels are also elevated in patients with stable vascular disease.34 It therefore appears that, although sP-selectin levels are highest in patients with unstable coronary disease, they are also elevated in patients with stable coronary disease and in patients with other pathologic conditions.