Special articleAnalysis of relaxation in the evaluation of ventricular function of the heart
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Echocardiographic evaluation of systolic and diastolic function in patients with cardiac amyloidosis
2011, American Journal of CardiologyCitation Excerpt :The pathomechanisms contributing to the accelerated diastolic dysfunction in CA are still under discussion. In early CA with mild amyloid infiltration, the relaxation process is altered by calcium overload.46–48 Gradually, as the degree of amyloid deposition increases, the left ventricle becomes stiffer, and thus increased filling pressure induces shortening of the isovolumetric relaxation time and generates the well-described pseudonormal diastolic filling pattern.36
Exercise Intolerance
2011, Cardiology ClinicsCitation Excerpt :In laboratory animal models calcium antagonists, particularly dihydropyridines, prevent ischemia-induced increases in LV diastolic stiffness87 and improve diastolic performance in pacing-induced heart failure.88–90 Negative inotropic calcium antagonists significantly impair early relaxation,90–94 however, and in general have shown a tendency toward adverse outcome in patients who have SHF.90 Nonetheless, Setaro and colleagues95 examined 22 men (mean age, 65 years) who had clinical heart failure despite an EF greater than 45% in a randomized, double-blind, placebo-controlled cross-over trial of verapamil.
Exercise Intolerance in Diastolic Heart Failure
2008, Diastology: Clinical Approach to Diastolic Heart FailureExercise Intolerance
2008, Heart Failure ClinicsCitation Excerpt :In laboratory animal models calcium antagonists, particularly dihydropyridines, prevent ischemia-induced increases in LV diastolic stiffness [87] and improve diastolic performance in pacing-induced heart failure [88–90]. Negative inotropic calcium antagonists significantly impair early relaxation [90–94], however, and in general have shown a tendency toward adverse outcome in patients who have SHF [90]. Nonetheless, Setaro and colleagues [95] examined 22 men (mean age, 65 years) who had clinical heart failure despite an EF greater than 45% in a randomized, double-blind, placebo-controlled cross-over trial of verapamil.
Congestive heart failure in the elderly
2007, International Journal of GerontologyDiastolic Heart Failure in the Elderly
2007, Heart Failure ClinicsCitation Excerpt :In laboratory animal models calcium antagonists, particularly dihydropyridines, prevent ischemia-induced increases in LV diastolic stiffness [107] and improve diastolic performance in pacing-induced HF [108–110]. Negative inotropic calcium antagonists significantly impair early relaxation, however [110–114], and in general have shown a tendency toward adverse outcome in patients who have systolic HF [110]. Setaro and coworkers [115] examined 22 men (mean age 65) who had clinical HF despite EF greater than 45% in a randomized, double-blind, placebo-controlled crossover trial of verapamil.