Renal arterial stenosis as a cause of high output cardiac failure

doi:10.1016/0167-5273(89)90285-4

International Journal of Cardiology

Volume 22, Issue 3, March 1989, Pages 404-406

https://doi.org/10.1016/0167-5273(89)90285-4 Get rights and content

Abstract

A patient is described in whom renal arterial stenosis in a functionally solitary kidney led to arterial hypertension and high output cardiac failure. The pulmonary oedema was resistant to conventional drug therapy but showed prompt regression after percutaneous transluminal angioplasty. This case report demonstrates the usefulness of cardiac output measurement in selected patients presenting with hypertension and pulmonary oedema.

References (4)

JH Laragh
Renovascular hypertension: a paradigm for all hypertension
J Hypertension
(1986)
TG Pickering
Medical management of renovascular hypertension

There are more references available in the full text version of this article.

Cited by (12)

A study on two-layered model (CassonNewtonian) for blood flow through an arterial stenosis: Axially variable slip velocity at the wall
2011, Journal of the Franklin Institute
Citation Excerpt :
This rheological information of blood in turn could be exploited for the development of new diagnostic tools for many diseases such as myocardial infarction, hypertension, renal, retinal, etc. [11,23,18].
The present paper sheds some light on a mathematical model for blood flow through stenosed arteries with axially variable peripheral layer thickness and variable slip at the wall. The model consists of a core region of suspension of all the erythrocytes assumed to be a Casson fluid and a peripheral layer of plasma as a Newtonian fluid. For such models, in literature, the peripheral layer thickness and slip velocity are assumed a priori based on experimental observations. In the present analysis, new analytic expressions for the thickness of the peripheral layer, slip velocity and core viscosity have been obtained in terms of measurable quantities (flow rate (Q), centerline velocity (U), pressure gradient (−dp/dz), plasma viscosity (μ_p) and yield stress (θ)). Using the experimental values of Q, U, (−dp/dz), μ_p and θ, the values of the peripheral layer thickness, core viscosity, and slip velocity at the wall have been computed. The theoretically obtained peripheral layer thickness has been compared with its experimental value. It is found that the agreement between the two is very good (error<1.4%). Further, a comparison between theoretical and experimental values of core viscosity is made and it is observed that the error between the two becomes 3.7465% in the case of two-layered model (Casson–Newtonian) for tube diameter 40 μm. The analysis developed here could be used to determine the more accurate values of the apparent viscosity of blood, agreeability, rigidity and deformability of red cells. This information of blood could be useful in the development of new diagnosis tools for many diseases.
The heart in renovascular disease-An association demanding further investigation
2006, International Journal of Cardiology
A close relationship exists between cardiovascular and renal disease; they often occur concomitantly, and abnormalities in either system are pathophysiologically important in both causing disease and determining clinical outcome in the other. Whilst the main focus of the article relates to the adverse association between atherosclerotic renovascular disease (ARVD) and the cardiovascular system, it is important to briefly review relevant epidemiology.
Prevention of recurrent pulmonary edema in patients with bilateral renovascular disease through renal artery stent placement
1999, American Journal of Hypertension
Pulmonary edema and congestive heart failure (both referred to here as PE) have been reported to be complications of bilateral renal artery stenosis or unilateral stenosis in a solitary functioning kidney (both referred to as BRAS). The goals of this study were to determine whether a history of PE was more common in patients with BRAS than in those with unilateral stenosis and a normal contralateral kidney (URAS), and whether recurrent PE could be prevented by renal artery stent placement.
We evaluated 90 consecutive patients with renovascular disease who were treated with percutaneous renal artery stent placement. History and clinical follow-up were obtained through chart review and phone contact with referring physicians. Mean follow-up was 18.4 months after stent placement. Twenty-three of 56 (41%) subjects with BRAS had a history of PE before revascularization, compared with four of 34 (12%) subjects with URAS (P = .05). Twenty-five of the 27 patients with history of PE had adequate clinical follow-up. Seventeen of the 22 (77%) subjects with BRAS and history of PE had no further PE after stent placement in one or both renal arteries. The five BRAS subjects with recurrent PE after stent placement had evidence of stent thrombosis or restenosis. In contrast, only one of three (33%) URAS subjects with a history of PE remained free of PE after stent placement. We conclude that PE is a common complication of BRAS, but not of URAS. In patients with BRAS, recurrent PE can be prevented by successful stent placement in one or both renal arteries.
Two-Fluid Model for Blood Flow Through a Tapered Arterial Stenosis: Effect of Non-zero Couple Stress Boundary Condition at the Interface
2017, International Journal of Applied and Computational Mathematics
Cardiorenal syndromes: Renal artery disease and congestive heart failure
2013, Renal Vascular Disease
The efficacy of renal angioplasty in patients with renal artery stenosis and flash oedema or congestive heart failure: A systematic review
2012, European Journal of Heart Failure

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Brief reportRenal arterial stenosis as a cause of high output cardiac failure

Abstract

Renovascular hypertension: a paradigm for all hypertension

J Hypertension

Medical management of renovascular hypertension

Brief report
Renal arterial stenosis as a cause of high output cardiac failure