Relevance of electrocardiographic findings, heart failure, and infarct site in assessing risk and timing of left ventricular free wall rupture during acute myocardial infarction
References (22)
- et al.
Cardiac rupture—challenge in diagnosis and management
Am J Cardiol
(1977) - et al.
Frequency of rupture of the left ventricular free wall or ventricular septum among necropsy cases of fatal acute myocardial infarction since introduction of coronary care units
Am J Cardiol
(1989) - et al.
Ventricular septal and free wall rupture complicating acute myocardial infarction. Experience in the Multicenter Investigation of Limitation of Infarct Size
Am Heart J
(1989) - et al.
Relation of left ventricular free wall rupture in acute myocardial infarction to forced immobilization
Am J Cardiol
(1988) - et al.
Rupture of the left ventricular free wall during acute myocardial infarction: analysis of 138 necropsy patients and comparison with 50 necropsy patients with acute myocardial infarctions without rupture
Am J Cardiol
(1988) - et al.
Association of eosinophils with cardiac rupture
Hum Pathol
(1985) - et al.
Cardiac rupture, a clinically predictable complication of acute myocardial infarction: report of 70 cases with clinicopathologic correlations
J Am Coll Cardiol
(1993) - et al.
Cardiac rupture in patients with acute myocardial infarction
Chest
(1987) - et al.
The spectrum of death after myocardial infarction: a necropsy study
Am Heart J
(1989) - et al.
Relation between use of antiinflammatory agents and left ventricular free wall rupture during acute myocardial infarction
Am J Cardiol
(1987)
Cited by (56)
Mechanical, inflammatory, and embolic complications of myocardial infarction: An emergency medicine review
2019, American Journal of Emergency MedicineCitation Excerpt :It is important to note, however, that the aforementioned findings vary with the location of infarction. The presence of an intraventricular conduction delay may be less prevalent in patients with VFWR, although the subsequent development of a new conduction delay may portend increased risk for rupture [28,36,40]. Echocardiography will demonstrate a pericardial effusion with signs of cardiac tamponade, including diastolic right ventricular collapse (high specificity), systolic right atrial collapse (earliest sign), a plethoric inferior vena cava with minimal respiratory variation (high sensitivity), and exaggerated respiratory cycle changes in mitral and tricuspid valve in-flow velocities as a surrogate for pulsus paradoxus [4,48].
Cardiac rupture in tako-tsubo cardiomyopathy with persistent ST-segment elevation
2012, International Journal of CardiologyPost-infarct cardiac rupture: Recent insights on pathogenesis and therapeutic interventions
2012, Pharmacology and TherapeuticsCitation Excerpt :However, when evaluating the clinical trend of a decline in rupture prevalence, one needs to be aware of the current extremely low rate of autopsy, which appears to be the only reliable way of confirming rupture diagnosis. In this regard, the percentage of autopsy-proven rupture out of total autopsied patients who died of acute MI has actually increased from under 10% during 1980s (Reddy & Roberts, 1989; Batts et al., 1990) to 12–65% in the recent decades, indicating the lack of effective strategies for rupture prevention (Figueras et al., 1995; Becker et al., 1999; Hutchins et al., 2002; Pouleur et al., 2010). In VALIANT trial, 24% of post-MI deaths were due to cardiac rupture confirmed by autopsy (Shamshad et al., 2010).
Usefulness of leads V7, V8, and V9 ST elevation to diagnose isolated posterior myocardial infarction
2011, International Journal of CardiologyInfarct size and post-infarct inflammation determine the risk of cardiac rupture in mice
2010, International Journal of CardiologyCitation Excerpt :Patients who died of rupture were more likely to have sustained hypertension, mental stress or physical strain, and usually had preserved LV function [3]. Figueras et al. [24] reported that the incidence of HF was lower in patients with than without subsequent rupture (12% vs. 84%). In addition, the ISIS-1 Trial showed that the β-antagonist atenolol reduced the risk of rupture implying a pro-rupture effect of the sympathoadrenergic activation, probably through hemodynamic enhancement [31].
Left Ventricular Free Wall Rupture After Acute Myocardial Infarction and Thrombolysis
2010, Dynamic Echocardiography