Exercise-induced hypertension in the arms due to impaired arterial reactivity after successful coarctation resection
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Cited by (41)
Correlation of exercise response in repaired coarctation of the aorta to left ventricular mass and geometry
2013, American Journal of CardiologyUsefulness of exercise-induced hypertension as predictor of chronic hypertension in adults after operative therapy for aortic isthmic coarctation in childhood
2011, American Journal of CardiologyCitation Excerpt :It is thought that exercise-induced hypertension in patients with CoA is caused by diminished arterial wall compliance and increased rigidity owing to the structural abnormal precoarctational wall, with more collagen and less smooth muscle tissue.16 Furthermore, it is hypothesized that the abnormal tissue in the precoarctational wall influences baroreceptor activity in the upper vascular bed, which might explain the pre- and postoperative hypertension.6,17–20 The prognostic significance of exercise-induced hypertension in patients with CoA is unknown.
Severe hypertensive response to exercise in patients with corrected coarctation of the aorta and normal baseline blood pressure
2011, Hipertension y Riesgo VascularExercise and Athletics in Adults with Congenital Heart Disease
2008, Congenital Heart Disease in AdultsAortic Arch Geometry and Exercise-Induced Hypertension in Aortic Coarctation
2007, American Journal of CardiologyVascular Remodeling After "Successful" Repair of Coarctation. Impact of Aortic Arch Geometry
2007, Journal of the American College of CardiologyCitation Excerpt :Hence, there is a rationale for understanding the mechanisms of increased IMT and stiffness after CoA repair. Abnormal findings in the vascular reactivity measures (FMD and GTN) are not consistently found in subjects who had CoA repair (4,9,10,24,25) and, indeed, there was a wide range of values in these parameters in the current study. Similarly, although long-term follow-up studies show a high prevalence of hypertension, a significant proportion of CoA survivors remain normotensive after 30 years (1,2,22).