ECG in clinical research and drugs developmentRadiofrequency catheter ablation for incessant atrioventricular nodal reentrant tachycardia normalized H-V block associated with tachycardia-induced cardiomyopathy
Section snippets
Case
A 30-year-old man had a history of palpitation attacks which occurred several times per year and terminated within a few minutes. No cardiac abnormality had ever been identified during annual physical examinations. Three weeks before admission to our hospital, the frequency of his palpitation attack increased and he complained of coincident dyspnea and fainting, upon admission to another hospital. An electrocardiogram displayed supraventricular tachycardia at a rate of 200 beats/min. He was
Discussion
A state resembling dilated cardiomyopathy can be induced by atrial fibrillation with rapid ventricular responses 3, 5, 7, atrial tachycardia 8, 9, accessory pathway reciprocating tachycardias (6), and AVNRT3, 10. Termination of the tachycardia leads to a regression of myocardial dysfunction. The precise mechanisms responsible for the so-called tachycardia-induced cardiomyopathy have not been elucidated. Hypotheses include depletion of myocardial high-energy phosphate stores 1, 11, altered
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Cited by (15)
Clinical and Electrophysiological Characteristics of Incessant Atrioventricular Nodal Re-Entrant Tachycardia
2016, JACC: Clinical ElectrophysiologyIncessant Atrioventricular Nodal Re-Entrant Tachycardia: What's So Different?
2016, JACC: Clinical ElectrophysiologyIncessant atrioventricular nodal reentrant tachycardia with tachycardia-induced cardiomyopathy, biventricular thrombosis, and pulmonary emboli
2016, HeartRhythm Case ReportsCitation Excerpt :To our knowledge, this is the first report of typical AVNRT provoking tachycardia-induced cardiomyopathy in an otherwise healthy individual and the first description of AVNRT associated with intracardiac thrombosis and pulmonary emboli. Although supraventricular tachycardia typically presents with palpitations and shortness of breath, heart rates ≥170 beats/min are more likely to be associated with disabling symptoms such as syncope and dizziness.3–6 Since AVNRT is almost always associated with palpitations, is often self-remitting, and rarely goes unrecognized for weeks on end, tachycardia-induced ventricular dilation and dysfunction appear to be exceedingly rare.
Tachycardia-Mediated Cardiomyopathy Secondary to Focal Atrial Tachycardia. Long-Term Outcome After Catheter Ablation
2009, Journal of the American College of CardiologyCitation Excerpt :In addition, children with permanent junctional reciprocating tachycardia appear vulnerable to the development of LV dysfunction, with 24 of 85 (28%) children affected in 1 series (21). TCM has also rarely been reported to complicate other frequent paroxysmal or persistent supraventricular tachycardias, including AV nodal re-entry tachycardia and AV re-entrant tachycardias (1,16,22). The diagnosis of AT may be challenging, particularly at ventricular rates of 110 beats/min as presented in the current study.