Hypothesis: apoptosis may be a mechanism for the transition to heart failure with chronic pressure overload

J Mol Cell Cardiol. 1994 Aug;26(8):943-8. doi: 10.1006/jmcc.1994.1115.
No abstract available

Publication types

  • Editorial
  • Research Support, U.S. Gov't, Non-P.H.S.

MeSH terms

  • Animals
  • Apoptosis* / genetics
  • Calcium / physiology
  • Cardiomegaly / complications*
  • Cardiomegaly / pathology
  • Cell Death
  • Heart Failure / etiology*
  • Heart Failure / pathology
  • Humans
  • Models, Cardiovascular*
  • Myocardium / pathology
  • Necrosis
  • Ventricular Pressure*

Substances

  • Calcium