Autoimmune and inflammatory responses may have an additive effect in postpercutaneous transluminal coronary angioplasty restenosis

A Blum; N Vardinon; G Kaplan; S Laniado; I Yust; M Burk; H Miller

doi:10.1016/s0002-9149(97)00914-4

Autoimmune and inflammatory responses may have an additive effect in postpercutaneous transluminal coronary angioplasty restenosis

Am J Cardiol. 1998 Feb 1;81(3):339-41. doi: 10.1016/s0002-9149(97)00914-4.

Authors

A Blum¹, N Vardinon, G Kaplan, S Laniado, I Yust, M Burk, H Miller

Affiliation

¹ Department of Cardiology, Tel-Aviv Medical Center, Sackler Medical School, Tel-Aviv University, Israel.

PMID: 9468079
DOI: 10.1016/s0002-9149(97)00914-4

Abstract

Patients who had an increase in their serum amyloid type A level of > 100% in the first 24 hours after percutaneous transluminal coronary angioplasty (PTCA) and also developed a positive antibody result (antinuclear factor or anticardiolipin), had a relative risk of 10.6 for developing restenosis in the first year after PTCA.

MeSH terms

Angina, Unstable / immunology*
Angina, Unstable / therapy
Angioplasty, Balloon, Coronary*
Antibody Formation
Autoantibodies
Autoimmunity*
Constriction, Pathologic
Female
Humans
Inflammation / immunology*
Male
Middle Aged
Prospective Studies
Recurrence
Serum Amyloid A Protein / analysis

Substances

Autoantibodies
Serum Amyloid A Protein