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Correspondence
Tumour necrosis factor-α is a foe for patients with acute myocardial infarction
  1. Kai-Hu Shi1,2,
  2. Hui Tao1,2,
  3. Jun-Xu Wu1,2
  1. 1 Department of Cardiothoracic Surgery, The Second Hospital of Anhui Medical University, Hefei, China
  2. 2 Cardiovascular Research Center, Anhui Medical University, Hefei, China
  1. Correspondence to Dr Kai-Hu Shi, Department of Cardiothoracic Surgery, The Second Hospital of Anhui Medical University, Furong Road, Anhui Province, Hefei 230601, China; ayskh3{at}hotmail.com

http://dx.doi.org/10.1136/heartjnl-2013-304199

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    To the Editor Tumour necrosis factor-α (TNF-α) has been implicated in the development of acute myocardial infarction. The mechanisms of TNF-α on endothelial function and platelet activation in patients with acute myocardial infarction are still unclear. A recent article by Padfield et al 1 demonstrated that TNF-α antagonism is unlikely to be a beneficial therapeutic strategy in patients with acute myocardial infarction. And plasma TNF-α concentrations increased in all patients following etanercept. This …

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    Footnotes

    • KHS and HT contributed equally to this letter.

    • Funding This project was supported by Anhui Provincial Natural Science Foundation (1308085MH117).

    • Provenance and peer review Not commissioned; internally peer reviewed.

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