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Low pulmonary artery size might be associated with subclinical systemic ventricular dysfunction in the Fontan circulation
  1. Kenan Yalta1,
  2. Orkide Palabiyik2,
  3. Ertan Yetkin3
  1. 1 Cardiology Department, Trakya University, Edirne, Turkey
  2. 2 Department of Biophysics, Trakya University, Edirne, Turkey
  3. 3 Cardiology Department, Istinye University, Liv Hospital, Istanbul, Turkey
  1. Correspondence to Kenan Yalta, Cardiology Department, Trakya University, Edirne 22030, Turkey; kyalta{at}

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Persistent symptoms and long-term complications in the setting of Fontan circulation (FC) (systemic ventricular failure, etc) still remain as significant clinical challenges in clinical practice.1 In their recently published interesting article,2 Ridderbos et al have suggested pulmonary artery (PA) size (as measured with Nakata index) as an independent predictor for functional clinical status (defined as peak oxygen consumption) in Fontan patients. The authors should be congratulated for the design and novel findings of the study. However, we would …

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  • Competing interests None declared.

  • Patient consent for publication Not required.

  • Provenance and peer review Not commissioned; internally peer reviewed

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