Atheromatous coronary stenoses are no longer considered to be passive structures, instead having the capacity for dynamic, often transient, change which may take the form of constriction or dilatation in response to either endogenous or external stimulation.
- nitric oxide
- coronary artery disease
- iNOS, cytokine induced nitric oxide synthase
- LNMMA, n-monomethyl l-arginine
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