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Vasomotion and nitric oxide bioactivity in diseased coronary arteries
  1. D Tousoulis1,
  2. G J Davies2,
  3. T Crake3,
  4. P C Toutouzas1
  1. 1Cardiology Unit, Athens University Medical School, Athens, Greece
  2. 2Division of Cardiology, Hammersmith Hospital, London, UK
  3. 3Department of Cardiology, St Bartholemew's Hospital, London, UK
  1. Correspondence to:
    Graham J Davies, Division of Cardiology, Hammersmith Hospital, Du Cane Road, London W12 ONN, UK;
    gjdavies{at}hhnt.org

Abstract

Atheromatous coronary stenoses are no longer considered to be passive structures, instead having the capacity for dynamic, often transient, change which may take the form of constriction or dilatation in response to either endogenous or external stimulation.

  • vasomotion
  • nitric oxide
  • coronary artery disease
  • iNOS, cytokine induced nitric oxide synthase
  • LNMMA, n-monomethyl l-arginine

https://doi.org/10.1136/heart.87.4.320

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