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C reactive protein for risk stratification in acute coronary syndromes? Verdict: unproven
  1. S Kennon1,
  2. A D Timmis1,
  3. R Whitbourn2,
  4. C Knight1
  1. 1Department of Cardiology, Bart’s and the London NHS Trust, London, UK
  2. 2Department of Cardiology, St Vincent’s Hospital, Melbourne, Australia
  1. Correspondence to:
    Dr Simon Kennon, Department of Cardiology, London Chest Hospital, Bonner Road, London E2 9JX, UK;
    srok{at}dircon.co.uk

Abstract

Although C reactive protein is intimately involved with the pathogenic mechanisms that drive acute coronary syndromes, there is no evidence that it is helpful for identifying patient groups who might benefit from particular treatment strategies

  • C reactive protein
  • inflammation
  • acute coronary syndromes
  • troponin
  • CAPTURE, chimeric 7E3 antiplatelet therapy in unstable angina refractory to standard treatment
  • CRP, C reactive protein
  • FRISC, Fragmin during instability in coronary artery disease
  • IL, interleukin
  • TIMI, thrombolysis in myocardial infarction

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