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Inflammatory and thrombotic mechanisms in coronary atherosclerosis
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  • Published on:
    Authors' Reply: Smoking and inflammation
    • Dimitris Tousoulis, Associate Professor
    • Other Contributors:
      • Charalambos Antoniades and Christodoulos Stefanadis

    Dear Editor

    In a response letter Cope et al. stated that smoking or the amount of tobacco consumed did not influence the biochemical risk factors for coronary artery disease such as cholesterol and HDL, but increased many of the cellular factors which we have reported in the recent review article as contributors to the inflammatory response and to the formation and instability of the atheromatous plaque.[1]...

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    Conflict of Interest:
    None declared.
  • Published on:
    Smoking and thrombotic mechanisms
    • Graham F. Cope, Honorary Senior Research Fellow
    • Other Contributors:
      • Nick Battersby, Medical Student

    Dear Editor

    We read the review paper by Tousoulis et al. with great interest. We applaud their thoroughness in describing the molecular and cellular mechanisms involved in the inflammatory mechanisms and haemostatic factors leading to coronary atherosclerosis. They also describe the role of chronic infection in this process.[1] However, they omit an important factor that is common to the majority of patients su...

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    Conflict of Interest:
    None declared.