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Correspondence
Response: Raised troponin levels in COPD: a possible mechanism
  1. Anke Neukamm1,2,
  2. Vidar Søyseth1,2,
  3. Torbjørn Omland1,2
  1. 1 Division of Medicine, Akershus University Hospital, Lørenskog, Norway
  2. 2 Institute of Clinical Medicine, University of Oslo, Lørenskog, Norway
  1. Correspondence to Professor Torbjørn Omland, Division of Medicine, Akershus University Hospital, and Institute of Clinical Medicine, University of Oslo, Lørenskog 1478, Norway; torbjorn.omland{at}medisin.uio.no

https://doi.org/10.1136/heartjnl-2013-303662

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    The Authors' reply We appreciate Dr Orde’s interest in potential mechanisms underlying the raised circulating troponin levels in COPD.1–4 Based on his own work,4 Dr Orde speculates that right ventricular myocardial necrosis and inflammation may represent a possible mechanism for troponin rise in stable COPD and COPD exacerbations. The study by Orde et …

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    Footnotes

    • Contributors All authors contributed equally to this paper.

    • Competing interests None.

    • Provenance and peer review Commissioned; internally peer reviewed.

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