@article {Henein389, author = {M Y Henein and R D Rakhit and M N Sheppard and D G Gibson}, title = {Restrictive pericarditis}, volume = {82}, number = {3}, pages = {389--392}, year = {1999}, doi = {10.1136/hrt.82.3.389}, publisher = {BMJ Publishing Group Ltd}, abstract = {BACKGROUND Pericardial thickening is an uncommon complication of cardiac surgery.OBJECTIVES To study pericardial thickening as the cause of severe postoperative venous congestion.SUBJECTS Two men, one with severe aortic stenosis and single coronary artery disease, and one with coronary artery disease after an old inferior infarction. Both had coronary artery bypass grafting surgery.METHODS Magnetic resonance imaging (MRI), Doppler echocardiography, and cardiac catheterisation.RESULTS Venous pressure was raised in both patients. MRI showed mildly thickened pericardium, and cardiac catheterisation indicated diastolic equalisation of pressures in the four chambers. Jugular venous pulse showed a dominant {\textquotedblleft}Y{\textquotedblright} descent coinciding with early diastolic flow in the superior vena cava, and mitral and tricuspid Doppler forward flow proved restrictive physiology. The clinical background suggested pericardial disease so both patients had pericardiectomy. This proved the pericardium to be thickened; the extent of fibrosis also involved the epicardium.CONCLUSIONS Although rare, restrictive pericarditis (restrictive ventricular physiology resulting from pericardial disease) should be considered to be a separate diagnostic entity because its pathological basis and treatment are different from intrinsic myocardial disease. This diagnosis may be confirmed by standard investigational techniques or may require diagnostic thoracotomy.}, issn = {1355-6037}, URL = {https://heart.bmj.com/content/82/3/389}, eprint = {https://heart.bmj.com/content/82/3/389.full.pdf}, journal = {Heart} }