Clinical communicationBlood plasma catecholamines and their urinary excretion in patients with acute myocardial infarction
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Cited by (27)
Hyperglycemia in nondiabetic patients presenting with acute myocardial infarction
2012, American Journal of the Medical SciencesCitation Excerpt :Thus, up to 100,000 admissions of nondiabetic patients with AMI in the United States are complicated by hyperglycemia yearly. Hyperglycemia during AMI may be due to stress with increased release of catecholamines,13–15 steroids,16–18 and glucagon19,20 and decreased release of insulin.21,22 Insulin deficiency is relative but is also the result of partial inhibition of pancreatic beta-cells by a stress-induced rise in catecholamines.23
Changes in vasoconstrictive hormones, natriuretic peptides, and left ventricular remodeling soon after anterior myocardial infarction
2001, American Heart JournalCitation Excerpt :The magnitude of the increase in plasma catecholamines observed in this study is also in agreement with previous studies. Pain and anxiety increase sympathetic discharge and increase neurohumoral activation globally.26,27 The effect of an ACE inhibitor on plasma catecholamines in the setting of an acute MI has been controversial.28-32
Activation of neurohumoral systems following acute myocardial infarction
1991, The American Journal of CardiologyPlasma catecholamines in acute myocardial infarction
1979, American Heart Journal