Postextrasystolic sinoatrial exit block in human sick sinus syndrome: Demonstration by direct recording of sinus node electrograms
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2005, Journal of the American College of CardiologyCitation Excerpt :Indeed, Scn5a-coded Na channels were distributed in the periphery of the mouse sinus node (24), and heterozygous Scn5aknockout mice showed a prolongation of the P wave as well as the PR interval, suggesting slow conduction within the atria and atrioventricular conduction system (25). Furthermore, a failure in impulse conduction in the adjacent atrial myocardium (exit block) is one of the pathophysiological bases of human SSS (26,27). Benson et al. (10) recently reported a young man with congenital SSS who had compound heterozygous SCN5Amutations, T220I and R1623X.
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The authors express their gratitude to Professor H. Warembourg, MD, and thank M. Blin, M. P. Théry, and I. Morenval for their expert secretarial assistance. We also thank Dr. E. Kraus for assistance with preparation of the English manuscript.