Diminished nocturnal decline in blood pressure in elderly hypertensive patients with left ventricular hypertrophy
Reference (26)
- et al.
Assessment of the daily blood pressure load as a determinant of cardiac function in patients with mild-to-moderate hypertension
Am Heart J
(1989) - et al.
Effects of chronic congestive heart failure secondary to coronary artery disease on the circadian rhythm of blood pressure and heart rate
Am J Cardiol
(1988) - et al.
Echocardiographic assessment of left ventricular hypertrophy: comparison to necropsy findings
Am J Cardiol
(1986) - et al.
Circadian variation of blood pressure
Lancet
(1978) - et al.
Relationship between ambulatory and exercise blood pressure and cardiac structure
Am Heart J
(1988) - et al.
Exercise systolic blood pressure: a powerful determinant of increased left ventricular mass in patients with hypertension
J Am Coll Cardiol
(1985) - et al.
Assessment of four ambulatory blood pressure monitors and measurements by clinicians versus in traarterial blood pressure at rest and during exercise
Am J Cardiol
(1990) - et al.
Population implications of electrocardiographic left ventricular hypertrophy
Am J Cardiol
(1987) - et al.
Value of echocardiographic measurement of left ventricular mass in predicting cardiovascular morbid events in hypertensive men
Ann Intern Med
(1986) - et al.
Average daily blood pressure, not office blood pressure, determines cardiac function in patients with hypertension
JAMA
(1989)
Relationship of 24-hour blood pressure mean and variability to severity of target-organ damage in hypertension
J Hypertens
Enhanced blood pressure response to regular daily stress in urban hypertensive men
J Hypertens
Lowest not highest blood pressure may determine left ventricular filling [Abstract]
Circulation
Cited by (131)
Regulation of angiotensinogen expression by angiotensin II in spontaneously hypertensive rat primary astrocyte cultures
2016, Brain ResearchCitation Excerpt :As the substrate of renin and the precursor of RAS peptides, the concentration of AGT is an important rate-determining molecule. Further, studies have shown that the expression of AGT is differentially regulated and cell type-specific (Brasier et al., 1989; Fukamizu et al., 1990; Kuwajima et al., 1992; McGehee et al., 1993). AGT exerts a pathogenic role in certain forms of hypertension, and aberrations of the AGT gene appear to co-segregate with a hypertensive phenotype (Jeunemaitre et al., 1992).
Ambulatory Blood Pressure Monitoring: Mercury Rising
2015, Advances in Chronic Kidney DiseaseFine particulate matter results in hemodynamic changes in subjects with blunted nocturnal blood pressure dipping
2014, Environmental ResearchCitation Excerpt :O׳Brien et al. (1998) introduced the dipping/non-dipping classification; the threshold value was a decrease of 10% in nocturnal blood pressure dipping. Such classification appears to have advantages from a clinical standpoint and has been used in several studies to demonstrate target organ damages and cardiovascular risks in non-dippers (i.e., those with nocturnal systolic blood pressure dipping of <10%) (Bianchi et al., 1994; Kuwajima et al., 1992; Ohkubo et al., 2002; Redon et al., 1994; Rizzoni et al., 1992; Timio et al., 1994; Verdecchia et al., 2012, 1990; Viera et al., 2012). Whether the non-dippers are more susceptible to particulate matter-related cardiovascular risks, however, remains unknown.
Cystatin c and blood pressure: Results of 24 h ambulatory blood pressure monitoring
2010, European Journal of Internal MedicineCitation Excerpt :Nocturnal blood pressure levels are independently associated with end-organ damage, over and above the risk associated with daytime values. It has also been shown that absence of nocturnal ‘dipping’ of blood pressure to lower levels than during the day is associated with target organ involvement [17,29–31]. ABPM is the only non-invasive blood pressure measuring technique that permits measurement of blood pressure during sleep.
Role of sleep-wake cycle on blood pressure circadian rhythms and hypertension
2007, Sleep MedicineCitation Excerpt :Absence of the normal 10–20% decline in BP during sleep appears to carry a higher heart and cerebral risk since the amount of time during the 24 h when BP is elevated and impacts target tissues and organs is prolonged. Accordingly, the average sleep-time BP level and the magnitude of the sleep-time BP decline are significantly correlated with target organ damage to the heart [36,177–182], brain [9,15,183,184], and kidney [185–189]. A large prospective study demonstrated that CV morbidity is higher in non-dipper than dipper hypertensive women [190].