Mechanism of mitral valve area increase by in vitro single and double balloon mitral valvotomy
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Cited by (65)
Percutaneous Balloon Mitral Valvuloplasty: Echocardiographic Eligibility and Procedural Guidance
2018, Interventional Cardiology ClinicsCitation Excerpt :Both the Wilkins and Padial scoring systems rely on the qualitative and semiquantitative assessment of mitral valve morphologic features, which are subject to interobserver variability and are less reliable for predicting success with scores in the mid-range. In vitro studies have demonstrated that the mechanism by which PBMV helps resolve rheumatic mitral stenosis is by splitting the fused commissures.17 As such, a valve with a greater extent of commissural fusion may benefit more from PBMV than a valve without any commissural fusion, in which the stenosis is due to rigid leaflets or annular narrowing.
Does the mitral valve recoil after percutaneous balloon valvotomy?
2011, Cardiovascular Revascularization MedicineCitation Excerpt :This suggests that passive stretching of the valve structures may not contribute significantly to the immediate gain in valve area seen after PTMC. The primary mechanism of relief of stenosis after balloon valvotomy is the splitting of fused commissures [5–10]. Several investigators [7,11] have also highlighted the contribution of passive stretching of the valve structures in determining the ultimate valve area.
Mitral Valve
2010, Dynamic EchocardiographyDelayed papillary muscle rupture following percutaneous mitral balloon valvotomy
2008, International Journal of CardiologyCitation Excerpt :Severe mitral regurgitation is a recognized complication of Inoue balloon mitral valvuloplasty, occurring in approximately 7.5% (range 2 to 19%) of procedures [2–4]. The mechanism of severe mitral regurgitation with the Inoue balloon has been leaflet or chorda tendinae rupture [3,4] and less frequently subvalvar apparatus damage including papillary muscle rupture [2]. Padial et al. [4] have identified leaflet rapture (with the anterior leaflet involved in 54% of the cases) as the most frequent mechanism of post-valvotomy regurgitation.
Predictors of long-term event-free survival and of freedom from restenosis after percutaneous balloon mitral commissurotomy
2001, American Heart JournalCitation Excerpt :After BMC, splitting of the fused commissures is the mechanism by which MVA is increased,25-27 which occurs even in presence of severely calcified valves. Moreover, in vitro analysis of surgically excised mitral valves26,27 showed that commissural splitting occurs more often in calcified MS as opposed to no calcified MS. Thus BMC often seemed to us to be a reasonable treatment in our population, even when the echocardiographic score was ≥12. The studies by Pan et al,7 Meneveau et al,11 and Iung et al14 showed a lower rate of event-free survival in patients with gross calcifications than in patients with extensive subvalvular disease.
Long-term clinical and echocardiographic outcome of percutaneous mitral valvuloplasty: Randomized comparison of Inoue and double-balloon techniques
2000, Journal of the American College of Cardiology