ArticleParasitic diseases of the heart I: Acute and chronic Chagas' disease
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Pericardial Disease in the Developing World
2023, Canadian Journal of CardiologyThe vasculature in chagas disease
2011, Advances in ParasitologyCitation Excerpt :Acutely infected rats developed changes in the endothelial layer characterized by EC swelling and a few points of cytoplasmic discontinuity that appeared as holes exposing the subendothelial collagen that is usually associated with platelet–fibrin aggregates, which might affect the generation of vasoactive substances, and impairs the equilibrium between opposing forces (Rossi, 1997). In vitro and in vivo studies indicate that infection of the endothelium results in expression of both pro-inflammatory cytokines and vascular adhesion molecules, which are important components of the inflammatory response (Huang et al., 1999a,b; Tanowitz et al., 1992a,b). Infection of ECs activates NF-κB and likely contributing to the induction of cytokine and adhesion molecular expression in the endothelium (Huang et al., 1999a).
The role of endothelin in the pathogenesis of Chagas' disease
2001, International Journal for ParasitologyCitation Excerpt :Vascular perfusion was also evaluated by autoradiographic imaging utilising the fatty acid analogue 19-ido-3,3-dimethyl-18 nonadecenoic acid (DMIVM). There was a marked decreased in heart perfusion during acute infection (Tanowitz et al., 1992b; Fig. 6). These studies demonstrated that T. cruzi infection caused a generalised vasculopathy in many vascular beds.
Fibrosis and inflammatory cells in human chronic chagasic myocarditis: Scanning electron microscopy and immunohistochemical observations
1998, International Journal of CardiologyPathogenesis of chronic Chagas' myocarditis: An overview
1996, Cardiovascular Pathology