Clinical investigationIncreased malondialdehyde in peripheral blood of patients with congestive heart failure☆
References (30)
Does reperfusion injury exist in humans?
J Am Coll Cardiol
(1993)- et al.
Free radical pathology in chronic arterial disease
Free Radic Biol Med
(1989) - et al.
Generation of free radicals and lipid peroxidation by redox cycling of Adriamycin and daunomycin
Biochem Biophys Res Commun
(1977) - et al.
Quinone-induced oxidative injury to cells and tissues
- et al.
An overview of contemporary etiologies of congestive heart failure
Am Heart J
(1991) - et al.
Evidence for enhanced free radical activity in chronic congestive heart failure secondary to coronary artery disease
Am J Cardiol
(1990) - et al.
Predictors of total mortality and sudden death in mild to moderate heart failure
J Am Coll Cardiol
(1989) - et al.
Lack of correlation between exercise capacity and indexes of resting left ventricular performance in heart failure
Am J Cardiol
(1981) Pathophysiology of chronic heart failure
Lancet
(1992)- et al.
The role of lipid peroxidation and antioxidants in oxidative modification of LDL
Free Radic Biol Med
(1992)
In vivo infusion of oxygen free radical substrates causes myocardial systolic but not diastolic dysfunction
Am Heart J
(1992)
Reactive oxygen species, neutrophil and endothelial adherence molecules, and lipid-derived inflammatory mediators in myocardial ischemia-reflow injury
Membrane lipid peroxidation induced by oxygen radical generation at reflow in reperfused heart
J Clin Invest
(1991)
Malondialdehyde in biological systems
Oxygen free radicals and congestive heart failure
Br Heart J
(1991)
Cited by (185)
Advances in congestive heart failure biomarkers
2023, Advances in Clinical ChemistryLipoxidation in cardiovascular diseases
2019, Redox BiologyVanillin mitigates potassium bromate-induced molecular, biochemical and histopathological changes in the kidney of adult mice
2016, Chemico-Biological InteractionsCitation Excerpt :The ability of the body to produce these antioxidants is controlled by genetic make-up and influenced by exposure to environmental factors [57]. More recently, a differential expression of SOD and GPx encoding genes has been used to detect biological toxicity and/or to monitor the impact of chemical pollutants [58,59]. The activities and genes expression of GPx and SOD increased in the KBrO3-treated mice.
- ☆
Partially supported by grants from Merck, Sharp & Dohme (I.A.) Corp., San Juan, Puerto Rico, and by the National Institutes of Health (grant No. RR03051, RCMI Program of the Medical Sciences Campus, University of Puerto Rico).
Copyright © 1996 Published by Mosby, Inc.