Clinical investigationRegional wall motion analysis by dobutamine stress echocardiography to distinguish between ischemic and nonischemic dilated cardiomyopathy
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Normal intima-media thickness on carotid ultrasound reliably excludes an ischemic cause of cardiomyopathy
2010, American Heart JournalCardiac Reserve in the Transplanted Heart: Effect of a Graft Polymorphism in the β<inf>1</inf>-adrenoceptor
2007, Journal of Heart and Lung TransplantationComparison of Regional Systolic Myocardial Velocities in Patients with Isolated Left Ventricular Noncompaction and Patients with Idiopathic Dilated Cardiomyopathy
2007, Journal of the American Society of EchocardiographyInfluence of Age, Hypertension, and Diabetes on Cardiac Reserve in a Rat Model
2007, Journal of the American Society of EchocardiographyCitation Excerpt :However, depressed catecholamine responsiveness and cardiac performance in streptozotocin-induced diabetic swine is caused by disturbed postreceptor signaling (seen as an increase in the Gi:Gs protein ratio) and not by down-regulation of β-adrenoceptor density.19 Low-dose DSE has been shown to be able to discriminate between ischemic and nonischemic cardiomyopathy,2 and to predict clinical outcome in patients with both idiopathic dilated cardiomyopathy and ischemia.3,20 Further, it has recently been demonstrated that DSE could predict LV dilatation and provide prognostic information in patients with acute myocardial infarction.21
Late gadolinium-enhanced cardiovascular magnetic resonance identifies patients with standardized definition of ischemic cardiomyopathy: A single centre experience
2007, International Journal of CardiologyCitation Excerpt :Absence of subendocardial LGE differentiated the great majority of patients whose behaviour would have indicated them as non-IC from a prognostic viewpoint (LV systolic dysfunction with no coronary disease, or disease in only one vessel without affecting the main coronary artery or the proximal segment of the anterior descending coronary artery) [5,6]. Other noninvasive methods have been used to distinguish among ischemic and idiopathic cardiomyopathy, including echocardiography [15–20], radionuclide ventriculography [21,22], stress myocardial perfusion imaging [23–26], positron emission tomography [27,28] and electron beam computer tomography [29]. The success of these different approaches to distinguish between both etiologies of cardiomyopathy has been variable.